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Study protocol schematic. ACL, anterior cruciate ligament; ALM, adenosine, lidocaine, and magnesium therapy; <t>CTX-II,</t> <t>C-terminal</t> cross-linked telopeptide of type II collagen; d, days; IA, intra-articular; IV, intravenous.
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Study protocol schematic. ACL, anterior cruciate ligament; ALM, adenosine, lidocaine, and magnesium therapy; <t>CTX-II,</t> <t>C-terminal</t> cross-linked telopeptide of type II collagen; d, days; IA, intra-articular; IV, intravenous.
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Study protocol schematic. ACL, anterior cruciate ligament; ALM, adenosine, lidocaine, and magnesium therapy; <t>CTX-II,</t> <t>C-terminal</t> cross-linked telopeptide of type II collagen; d, days; IA, intra-articular; IV, intravenous.
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Study protocol schematic. ACL, anterior cruciate ligament; ALM, adenosine, lidocaine, and magnesium therapy; <t>CTX-II,</t> <t>C-terminal</t> cross-linked telopeptide of type II collagen; d, days; IA, intra-articular; IV, intravenous.
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Study protocol schematic. ACL, anterior cruciate ligament; ALM, adenosine, lidocaine, and magnesium therapy; <t>CTX-II,</t> <t>C-terminal</t> cross-linked telopeptide of type II collagen; d, days; IA, intra-articular; IV, intravenous.
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Study protocol schematic. ACL, anterior cruciate ligament; ALM, adenosine, lidocaine, and magnesium therapy; <t>CTX-II,</t> <t>C-terminal</t> cross-linked telopeptide of type II collagen; d, days; IA, intra-articular; IV, intravenous.
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Study protocol schematic. ACL, anterior cruciate ligament; ALM, adenosine, lidocaine, and magnesium therapy; <t>CTX-II,</t> <t>C-terminal</t> cross-linked telopeptide of type II collagen; d, days; IA, intra-articular; IV, intravenous.
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Study protocol schematic. ACL, anterior cruciate ligament; ALM, adenosine, lidocaine, and magnesium therapy; <t>CTX-II,</t> <t>C-terminal</t> cross-linked telopeptide of type II collagen; d, days; IA, intra-articular; IV, intravenous.
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crosslinked c-telopeptides of type ii collagen (ctx-ii)  (MyBiosource Biotechnology)
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MyBiosource Biotechnology crosslinked c-telopeptides of type ii collagen (ctx-ii)
Study protocol schematic. ACL, anterior cruciate ligament; ALM, adenosine, lidocaine, and magnesium therapy; <t>CTX-II,</t> <t>C-terminal</t> cross-linked telopeptide of type II collagen; d, days; IA, intra-articular; IV, intravenous.
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crosslinked c-telopeptides of type ii collagen (ctx-ii) - by Bioz Stars, 2026-06
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Image Search Results


Study protocol schematic. ACL, anterior cruciate ligament; ALM, adenosine, lidocaine, and magnesium therapy; CTX-II, C-terminal cross-linked telopeptide of type II collagen; d, days; IA, intra-articular; IV, intravenous.

Journal: Bone & Joint Research

Article Title: Adenosine, lidocaine, and magnesium therapy augments joint tissue healing following experimental anterior cruciate ligament rupture and reconstruction

doi: 10.1302/2046-3758.136.BJR-2023-0360.R1

Figure Lengend Snippet: Study protocol schematic. ACL, anterior cruciate ligament; ALM, adenosine, lidocaine, and magnesium therapy; CTX-II, C-terminal cross-linked telopeptide of type II collagen; d, days; IA, intra-articular; IV, intravenous.

Article Snippet: C-terminal cross-linked telopeptide of type II collagen (CTX-II), a marker of cartilage degradation, was measured in urine collected at day -1, 3, 8, and 28 following surgery (Rat CTX-II ELISA kit, E-EL-R2554; Elabscience, USA), and normalized to urinary creatinine (Creatinine Urinary Detection Kit, EIACUN; Thermo Fisher Scientific, USA), according to manufacturer’s instructions.

Techniques:

Cartilage degenerative changes in male and female adenosine, lidocaine, and magnesium (ALM)-treated and Saline control animals, 28 days after anterior cruciate ligament reconstruction (ACLR) surgery. a) Postoperative urinary CTX-II levels peaked in the first postoperative week, and were higher in females than males. b) Osteoarthritis Research Society International (OARSI) scoring of medial articular cartilage surfaces, and c) representative safranin O/fast green-stained sections (magnification ×100) of medial femoral condyles (MFC, upper panels) and medial tibial plateau (MTP, lower panels). Minor proteoglycan loss (asterisk), and surface fibrillation (arrows) was evident, with no sex- or treatment-specific differences. d) Relative expression of cartilage extracellular matrix (ECM) components (collagen type 2, Col2a1; aggrecan, Acan), ECM remodelling enzymes (matrix metalloproteinase-13, MMP13; tissue inhibitor of matrix metalloproteinase 1, Timp1); and mediators of cell proliferation and activation (transforming growth factor β 1, Tgfb1; α smooth muscle actin, Acta2) in MFC cartilage. MMP13 levels were higher in males than females, with ALM dampening this response. In females, expression of Acan, Tgfb1, and Acta2 was increased in ALM-treated, than Saline control animals. e) Representative α-SMA-stained sections of MFC indicating the presence of positive-staining cells in the superficial and transitional zones (arrowheads) of ALM-treated animals. Data show medians and interquartile ranges. *p < 0.05, Kruskal–Wallis test.

Journal: Bone & Joint Research

Article Title: Adenosine, lidocaine, and magnesium therapy augments joint tissue healing following experimental anterior cruciate ligament rupture and reconstruction

doi: 10.1302/2046-3758.136.BJR-2023-0360.R1

Figure Lengend Snippet: Cartilage degenerative changes in male and female adenosine, lidocaine, and magnesium (ALM)-treated and Saline control animals, 28 days after anterior cruciate ligament reconstruction (ACLR) surgery. a) Postoperative urinary CTX-II levels peaked in the first postoperative week, and were higher in females than males. b) Osteoarthritis Research Society International (OARSI) scoring of medial articular cartilage surfaces, and c) representative safranin O/fast green-stained sections (magnification ×100) of medial femoral condyles (MFC, upper panels) and medial tibial plateau (MTP, lower panels). Minor proteoglycan loss (asterisk), and surface fibrillation (arrows) was evident, with no sex- or treatment-specific differences. d) Relative expression of cartilage extracellular matrix (ECM) components (collagen type 2, Col2a1; aggrecan, Acan), ECM remodelling enzymes (matrix metalloproteinase-13, MMP13; tissue inhibitor of matrix metalloproteinase 1, Timp1); and mediators of cell proliferation and activation (transforming growth factor β 1, Tgfb1; α smooth muscle actin, Acta2) in MFC cartilage. MMP13 levels were higher in males than females, with ALM dampening this response. In females, expression of Acan, Tgfb1, and Acta2 was increased in ALM-treated, than Saline control animals. e) Representative α-SMA-stained sections of MFC indicating the presence of positive-staining cells in the superficial and transitional zones (arrowheads) of ALM-treated animals. Data show medians and interquartile ranges. *p < 0.05, Kruskal–Wallis test.

Article Snippet: C-terminal cross-linked telopeptide of type II collagen (CTX-II), a marker of cartilage degradation, was measured in urine collected at day -1, 3, 8, and 28 following surgery (Rat CTX-II ELISA kit, E-EL-R2554; Elabscience, USA), and normalized to urinary creatinine (Creatinine Urinary Detection Kit, EIACUN; Thermo Fisher Scientific, USA), according to manufacturer’s instructions.

Techniques: Saline, Control, Staining, Expressing, Activation Assay